Recently, a study published in the international magazine Nature Communications pointed out that researchers from institutions such as the Westmead Research Institute have found that serum zinc may help the body to effectively resist the occurrence of liver diseases; the researchers said that natural conditions Zinc can help inhibit the inflammatory and antiviral effects of interferon λ3 (IFN-λ3), and protein interferon λ3 is directly related to tissue damage in patients with chronic liver disease.
Researcher Dr. Scott Read said that this discovery provided the first evidence for researchers that zinc can act as a potential inhibitor of IFN-λ3 in the context of viral infections (such as hepatitis C and influenza virus infections). Zinc can inhibit many aspects of the liver's response to viral immune responses mediated by IFN-λ3. In addition, zinc can interfere with the binding of IFN-λ3 to the interferon λ receptor, and interferon λ receptors are tested in vivo or in vitro. Can reduce the body's anti-virus activity and increase the replication of the virus.
More interestingly, the researchers also found that zinc can also block the inflammatory activity of IFN-λ3, and that IFN-λ3 is directly related to the accelerated progression of cirrhosis in viral and non-viral liver diseases. The data shows that the level of zinc in the body of patients with chronic hepatitis C can be genetically determined by the polymorphism of IFN-λ3, which confirms the inhibitory role of zinc in in vivo experiments.
Researcher Read summarized that in this study, zinc may be potentially used as a simple and effective therapy to help protect against the occurrence of acute and chronic liver inflammation. The scientific community is currently developing more intensive IFN-λ3-mediated studies. Therapeutic intervention strategies for chronic diseases.
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