Medicinal efficacy of purple sweet potato

Selenium and iron in purple sweet potato are essential elements in the human body for anti-fatigue, anti-aging, and blood-supplementation. In particular, selenium is known as the “anti-cancer king” and is easily absorbed by the body. It can stay in serum, repair the heart muscle, and enhance immunity. The elimination of free radicals in the body inhibits the synthesis of DNA in cancer cells and the division and growth of cancer cells, and prevents the occurrence of cancerous diseases such as gastric cancer and liver cancer.

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Purple sweet potato is rich in cellulose, which increases fecal volume, promotes gastrointestinal motility, cleans up the retained mucus, accumulated gas, and putrefaction in the intestine, discharges toxic substances and carcinogens in the feces, keeps the stool open, improves the digestive tract environment, and prevents Occurrence of gastrointestinal diseases. In addition to the nutritious components of ordinary sweet potatoes, it also contains polysaccharides and flavonoids, and it is also rich in selenium and anthocyanins, and has certain functions of preventing hypertension, reducing liver dysfunction, and preventing cancer.

Yoshimot et al evaluated the anti-mutagenicity of four different color potato water extracts with Salmonella typhimurium TA98. It was found that anthocyanin of purple sweet potato can effectively inhibit the mutation caused by heterocyclic amines. Two anthocyanins isolated from purple sweet potato have a strong antimutagenic effect.

Animal experiments show that purple sweet potato beverages containing a large amount of anthocyanins can significantly inhibit the serum glutamate induced by carbon tetrachloride in liver injury. The increase of acetic transaminase (GOT) and glutamic acid-pyrogluconate transaminase (GTP), and inhibited the increase of thiobarbituric acid (TBA) reactants and oxidized lipoproteins in serum and liver; The yellow-sweet sweet-potato beverages cannot be used by Fredenck et al. for a total of 5 days per week for 4 weeks. Rats were intragastrically administered 2.5 mL of 5% ethanol solution/kg body weight containing 1%, 5% and 10% anthocyanins, respectively; Rats were given intragastric administration of 0.5 mL carbon tetrachloride/kg body weight. After 18 hours, the serum aspartate and alanine transaminase levels were significantly lower than those in the control group given only intragastric administration of carbon tetrachloride, indicating that anthocyanin could be inhibited. Carbon tetrachloride induced acute liver injury.


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